Ben,
some more info on uric acid that may be of use?


How Can We Cure NO/ONOO− Cycle Diseases?
Approaches to Curing Chronic Fatigue Syndrome/Myalgic Encephalomyelitis, Fibromyalgia, Multiple Chemical Sensitivity, Gulf War Syndrome and Possibly Many Others by Martin L. Pall, PhD


Uric acid levels in the blood are often about 4 to 5 times those of ascorbate, although there is quite a bit of variation around those figures. However the effectiveness of uric acid in scavenging peroxynitrite and its products, per mole, is roughly similar to that of ascorbate.31


Consequently, even though it is possible to raise ascorbate levels by much higher percentages than uric acid levels in vivo, it seems likely that raising uric acid levels will produce a substantial effect on peroxynitrite-mediated oxidations in vivo and therefore should be considered well worth pursuing in lowering the central couplet.

Uric acid has a half-life of about 20 hours in humans, so it should not take very long to increase its levels by increasing the availability of purine-containing compounds in the body, such that when an increase in purine degradation is obtained, it will be sustained substantially longer than any high-level ascorbate elevation.53

Consequently, it makes sense to consider each of these three supplements – inosine, RNA, and D-ribose – as possible agents to raise uric acid levels.

A second, related issue is that very high uric acid levels may produce hypertension, and while direct measurements suggest that uric acid lowers nitric oxide synthase uncoupling, rather than raising it, this also suggests that we should limit the rise in uric acid levels in these treatments.

With these two caveats in mind, a substantial rise in uric acid levels into the mid-to upper-normal range may be very helpful to people suffering from NO/ONOO− cycle diseases.


http://www.townsendletter.com/FebMarch2010/cureNO0210.html

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Into the heart, an air that kills, from yon far country blows.
What are those blue remembered hills, what sphires what farms are those.
That is the land of lost content,I see it shining plain,
The happy highways where I went and cannot come again

Hello Olly

This link may be worth looking into.

http://www.iub.edu/~sengelab/research/spinal_motoneuron.shtml

I am currently experimenting with testosterone depo and nandrolone but don't have sufficient data to submit yet.

Merc

maybe Meaghers theory needs further investigation. I cannot find anything more about it but Olly you seem to be inquisitive online seeker, maybe you will? He claimed mnd to be sex hormones imbalance

meagher was underlying its not only testosterone but also FSH and LH. He treated himself with pergonal and somethin else, but I dont remember exacly and cant find. There was another guy who pursued this trace and supported his observation

To summarize Meagher's faulty assumptions and conclusion:
1. Testosterone is necessary for the life of motor neurons.
2. FSH is necessary to transport testosterone into motor neurons.
3. Therefore pituitary failure causes low FSH, which in turn causes an insufficient amount of testosterone to reach the motor neurons, which then causes their death.

My first reading is 5.4 mg/dL

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If it is done in secret, it is done in vain.

Those guys at PLM are awesome. I asked them to add Uric Acid to their list of "labs" and the responded quickly. We can now report our Uric Acid levels. I strongly urge all PALS to request a uric acid test order from their physician.

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If it is done in secret, it is done in vain.

HP.
Normal values of Uric acid are from 0.20-0,42 mmol/l in male and in female 0,12-0,34 mmol/l

Jan

That's good news. Keep us abreast of changes both good and bad. I will trade gout for this crapfest disease any day of the week

Members..
For your information the lab check my uric acid level and it is 0,23 mmol/l

Jan